The Aβ(1–38) peptide is a negative regulator of the Aβ(1–42) peptide implicated in Alzheimer disease progression | Scientific Reports
Aβ1-42 Induced Neurotoxicity Assay - Alzheimer's Disease in vitro Model
Brain Sciences | Free Full-Text | Prognostic Role of CSF β-amyloid 1–42/1–40 Ratio in Patients Affected by Amyotrophic Lateral Sclerosis
Figure 1 from Amyloid-beta(1-42) rapidly forms protofibrils and oligomers by distinct pathways in low concentrations of sodium dodecylsulfate. | Semantic Scholar
IJMS | Free Full-Text | Aggregation States of Aβ1–40, Aβ1–42 and Aβp3–42 Amyloid Beta Peptides: A SANS Study
Solution NMR Studies of the Aβ(1−40) and Aβ(1−42) Peptides Establish that the Met35 Oxidation State Affects the Mechanism of Amyloid Formation | Journal of the American Chemical Society
Treatment of beta amyloid 1–42 (Aβ1–42)-induced basal forebrain cholinergic damage by a non-classical estrogen signaling activator in vivo | Scientific Reports
Human Amyloid Beta 1-42 Oligomers (SPR-488_
Amyloid beta - Wikipedia
Amyloid beta 1–42-induced animal model of dementia: a review - ScienceDirect
β-Amyloid (1-42), human - GenScript
Frontiers | The Acute Effects of Amyloid-Beta1–42 on Glutamatergic Receptor and Transporter Expression in the Mouse Hippocampus
Brain Atrophy in Healthy Aging Is Related to CSF Levels of Ab1-42 - LCBC
The Aβ1–42/Aβ1–40 ratio in CSF is more strongly associated to tau markers and clinical progression than Aβ1–42 alone | Alzheimer's Research & Therapy | Full Text